Buying a short sale with an hoa lien - Zillow Real Estate Advice

Someone has to pay off the lien before title can transfer - period. If it is you or the bank, someone has to. The home owner surely has been sent requests for payment and there is probably a lien filed at the courthouse that is public knowledge, you just need to find it. Are you using a lawyer to close the deal? The owner should be able to contact the HOA, but it sounds like they are not lifting a finger to help == typical in a short sale where they don't care. Do more digging.

Tim

Source: http://www.zillow.com/advice-thread/Buying-a-short-sale-with-an-hoa-lien/461910/

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the tap: Aspartame kills. Robinsons barley water has it.

By Dr. Mercola

More than 90 countries have given the artificial sweetener aspartame the green light to be used in thousands of food and beverage products.1

Two hundred times sweeter than sugar, aspartame allows food manufacturers to produce sweet foods they can market as ?low calorie,? ?diet,? or sugar-free,? appealing to hundreds of millions of consumers looking to cut sugar from their diets.

No doubt about it, the less sugar you include in your diet, the better. But replacing sugar with aspartame is not the solution, and in fact is likely to be even?worse?for your health.

Despite assurances from the U.S. Food and Drug Administration (FDA) and other public health agencies that aspartame is safe, the research says otherwise?

So What the Heck is Aspartame Made Of?

Virtually all of the marketing material emphasizes the fact that aspartame is natural and made of two amino acids, the building blocks of protein. But, like many deceptions, this is only partially true. While there are two amino acids that comprise 90% of aspartame, aspartic acid and phenylalanine, they are held together in a methyl ester bond that comprises 10% of the molecule.

The methanol is released from the aspartame within hours of consumption after hydrolysis of the methyl group of the dipeptide by chymotrypsin in the small intestine. Once this methyl ester bond is broken it liberates free methyl alcohol or methanol, which is commonly called wood alcohol. The problem with methanol is that it passes into your blood-brain barrier and is converted into formaldehyde, which causes the damage. You may recognize formaldehyde as embalming fluid.

Interestingly, methanol is only toxic in humans. All other animals are able to detoxify it before it causes damage.

Methanol is a toxin that destroys the myelin tissue in your body, which is the insulating material around your nerves that allows nerve signals to travel properly. Once injured, one can have what are called demyelinating symptoms that are commonly seen in diseases like MS and also migraines?that can?include bizarre and inconsistent visual field disruptions.

My sister that helped me start my practice in 1985 is actually one of the people that develops these symptoms when exposed to aspartame. In the late ?80s I helped to diagnose her with this sensitivity and she has avoided it for over 25 years.

Why is Methanol So Toxic?

Methanol breaks down into formic acid and formaldehyde in your body. Many experts believe formic acid is the problem but the real problem is the formaldehyde, which is a deadly neurotoxin and carcinogen. An EPA assessment of methanol states that methanol "is considered a cumulative poison due to the low rate of excretion once it is absorbed. In the body, methanol is oxidized to formaldehyde and formic acid; both of these metabolites are toxic."2 They recommend a limit of consumption of 7.8 mg/day. But according to Woodrow Monte, Ph.D, R.D., director of the Food Science and Nutrition Laboratory at Arizona State University:3

?When diet sodas and soft drinks, sweetened with aspartame, are used to replace fluid loss during exercise and physical exertion in hot climates, the intake of methanol can exceed 250 mg/day or 32 times the Environmental Protection Agency's recommended limit of consumption for this cumulative toxin.?

Further, he states that due to the lack of a couple of key enzymes, humans are many times more sensitive to the toxic effects of methanol than animals. Therefore, tests of aspartame or methanol on animals do not accurately reflect the danger for humans.

?There are no human or mammalian studies to evaluate the possible mutagenic, teratogenic, or carcinogenic effects of chronic administration of methyl alcohol,??he said.

Symptoms from methanol poisoning are many, and include headaches, ear buzzing, dizziness, nausea, gastrointestinal disturbances, weakness, vertigo, chills, memory lapses, numbness and shooting pains in the extremities, behavioral disturbances, and neuritis. The most well known problems from methanol poisoning are vision problems including misty vision, progressive contraction of visual fields, blurring of vision, obscuration of vision, retinal damage, and blindness. Formaldehyde is a known carcinogen that causes retinal damage, interferes with DNA replication and may cause birth defects. The researchers in the featured study then reasoned that the aspartame-induced methanol exposure was likely possible for oxidative stress in the brain.

New Study Shows Aspartame Damages Your Brain

A newly published study with rats investigated the chronic effect of aspartame on oxidative stress in the brain. Researchers found that there was a significant increase in lipid peroxidation levels, superoxide dismutase activity, GPx levels and CAT activity, showing that chronic exposure of aspartame resulted in detectable methanol in the blood, which may be responsible for the generation of oxidative stress and damage in the brain.4

So the study found that aspartame exposure did result in ?detectable levels? of methanol in the blood. Methanol is gradually released in the small intestine when the methyl group of aspartame encounters the enzyme chymotrypsin.

Are Artificial Sweeteners Stressing Out Your Brain?

Oxidative stress can be defined as the state in which damaging free radicals outnumber your antioxidant defenses. Oxidative stress tends to lead to accelerated tissue and organ damage.

Case in point, earlier this year another study investigated the effect of long-term intake of aspartame on the antioxidant defense status in the rat brain and also found it leads to oxidative stress.5?Male rats that were given a high dose of the artificial sweetener exhibited a lowered concentration of reduced glutathione (the active, antioxidant form of glutathione), and reduced glutathione reductase activity, a sign of increased oxidative stress-induced damage in the body.

Glutathione deficiency has also been linked to age-related diseases such as Alzheimer's. Examination also revealed mild vascular congestion ? an obstruction of the normal flow of blood within the brain ? in these rats. Researchers concluded:

"The results of this experiment indicate that long-term consumption of aspartame leads to an imbalance in the antioxidant/pro-oxidant status in the brain, mainly through the mechanism involving the glutathione-dependent system."

Adding to the problem, one of the amino acids in aspartame, aspartic acid is capable of crossing your blood-brain barrier. There it attacks your brain cells, creating a form of cellular overstimulation called excitotoxicity, which can lead to cell death.

Your blood-brain barrier, which normally protects your brain from excess aspartate, as well as toxins, is not able to adequately protect you against the effects of aspartame consumption because it:

  • Is not fully developed during childhood
  • Does not fully protect all areas of the brain
  • Is damaged by numerous chronic and acute conditions
  • Allows seepage of excess aspartate into the brain even when intact

That excess aspartate slowly begins to destroy neurons, and the large majority (75 percent or more) of neural cells in a particular area of the brain are killed before any clinical symptoms of a chronic illness are noticed. Then, when they do occur, they may or may not be associated with aspartame consumption, even though examples of chronic illnesses that are made worse by long-term exposure to excitatory amino acid damage include:

Multiple sclerosis (MS)ALSMemory loss
Hormonal problemsHearing lossEpilepsy
Alzheimer's disease and dementiaParkinson's diseaseHypoglycemia
AIDSBrain lesionsNeuroendocrine disorders

Why Was Aspartame Ever Approved?

If it causes brain damage, why is aspartame allowed in our food and drinks? The truth of the matter is the FDA rejected aspartame not once but multiple times. The scientific data just did not support it as a safe product. But the FDA is a federal agency subject to the political winds, and the people in charge of the agency have repeatedly and notoriously been accused of many conflicts of interest, both economically and ethically.

In 1975, the FDA came to the conclusion that aspartame should not be allowed on the market. They requested that further studies be conducted. The FDA's next move was to set up a public board of inquiry composed of outside experts to investigate the safety of aspartame, and in 1980 that board unanimously?rejected?aspartame's request for approval. Another internal FDA panel convened in 1980 also rejected aspartame for approval.

So it was three strikes against aspartame at this point, four strikes if you count the Bressler Report. This report was compiled in 1977 after FDA scientists looked into the field studies conducted on aspartame. The Bressler Report uncovered fraud and manipulation of data so serious that the FDA forwarded their files to the Chicago U.S. Attorney's office for prosecution.

Basically the results of the scientific data were fairly clear up until 1980: Aspartame was a dangerous, brain-tumor-causing man-made poison and the company trying to get it into the food supply was recommended for prosecution by the FDA. You would think that would be the end of aspartame, right?

Not by a long shot.

For more details on the story of how aspartame made it through the FDA approval process despite warning signs of potential health hazards and alleged scientific fraud, please watch the 60-Minutes report below, as Mike Wallace does a nice job of summarizing an otherwise very?long story.

Did You Know Aspartame May Make You Fat?

If you?re one of the people who suffers from headaches/migraines, vision problems, fatigue, anxiety attacks,?abdominal pains or other symptoms when you consume aspartame, deciding to eliminate it?from your diet was probably an easy choice.

For the rest of you, doing so based on the possibility that it could ?one day? cause symptoms of brain damage is much more abstract, and probably much less likely to make you take action today.

That?s why I want to share with you one of the major deceptions surrounding artificial sweeteners like aspartame, which is that they will help you lose weight by avoiding sugar.

  • Stimulate your appetite
  • Increase carbohydrate cravings
  • Stimulate fat storage and weight gain. In fact, diet sodas, which are well-known sources of artificial sweeteners, may actually?double?your risk of obesity!6

So much for being a dieter's best friend... The point is, if you?re having a hard time giving up aspartame based on its potential to damage your brain, maybe the fact that it could make you pack on the pounds in the very near future will motivate you toward positive change.

My Favorite Tool for Addressing Artificial Sweetener Addictions

Artificial sweeteners tend to trigger enhanced activity within your brain's pleasure centers, yet at the same time provide less actual satisfaction. This separation of the taste of sweetness from caloric content means that when you consume artificial sweeteners, your brain actually craves more of it because your body receives no satisfaction on a cellular level by the sugar imposter. This can actually contribute to not only overeating and weight gain, but also an addiction to artificial sweeteners.

In order to break free, be sure you address the emotional component to your food cravings using a tool such as theEmotional Freedom Technique (EFT). More than any traditional or alternative method I have used or researched, EFT works to overcome food cravings and helps you reach dietary success. If diet soda is the culprit for you, be sure to check out?Turbo Tapping, which is an extremely effective and simple tool to get rid of your soda addiction in a short amount of time.

If you're determined to sweeten your foods and beverages, I urge you to consider using stevia extract ? a safe and natural sweet herb, which is my personal sweetener of choice. Lo Han is another herbal sweetener that doesn?t have the aftertaste of stevia that many object to.

Source: http://the-tap.blogspot.com/2012/09/aspartame-kills-robinsons-barley-water.html

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Guess Where? Monkey - Carry On | Travel Leisure

09.28.12

201103-b-guesswhere-145.jpg

Sponsored by Delta

Can you guess which country these monkey call home? I'll give you one hint. It's country that Delta, a parter of this weekend's Travel + Leisure's Global Bazaar, flies to.

Log in and leave your guesses below.

Check back on Monday for the answer!

Lyndsey Matthews is an assistant digital editor at Travel + Leisure. Follow her on Twitter @matthewslyndsey

Photo Courtesy of the T+L Photo Contest

Source: http://www.travelandleisure.com/travel-blog/carry-on/2012/9/28/guess-where-monkey

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Michelle Obama's Popular DNC Dress Out in December

DEAR ABBY: My husband and I just got some shocking news. His father -- age 81 -- is leaving his wife of 60 years! Mom is not entirely self-sufficient and seems dependent on him.Dad found himself a younger woman -- a "chick" of 70. He has announced that he still has sexual needs and wants to enjoy the rest of his life. My husband thinks it will be a short-term fling and he'll return to Mom, but she says she won't be taking him back. (Who knows how she'll feel later?)My problem is, no matter what happens between them, I'm having a hard time even considering forgiving him for his selfishness. ...

Source: http://news.yahoo.com/michelle-obamas-popular-dnc-dress-december-193759668--abc-news-politics.html

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Combination of targeted treatment drugs delays resistance in melanoma patients

[ Back to EurekAlert! ] Public release date: 29-Sep-2012
[ | E-mail | Share Share ]

Contact: Katie Marquedant
kmarquedant@partners.org
617-726-0337
Massachusetts General Hospital

Combined treatment with two drugs targeting different points in the same growth-factor pathway delayed the development of treatment resistance in patients with BRAF-positive metastatic malignant melanoma. The results of a phase I/II study of treatment with the kinase inhibitors dabrafenib and trametinib will be published in the New England Journal of Medicine and are being released online to coincide with a presentation at the European Society for Medical Oncology meeting in Vienna.

"We investigated this combination because of research we and others have conducted into the molecular underpinnings of resistance to BRAF inhibitor therapy," says Keith Flaherty, MD, of the Massachustts General Hospital (MGH) Cancer Center, lead author of the NEJM report and principal investigator of the study. "We found that adding the MEK inhibitor trametinib to BRAF inhibitor dabrafenib clearly delays the emergence of resistance. In fact, the combination was at least twice as effective as BRAF inhibition alone."

In around half of patients with metastatic melanoma, tumor growth is driven by mutations that keep the BRAF protein part of the MAPK cell growth pathway constantly activated. In recent years, drugs that inhibit BRAF activity have rapidly halted and reversed tumor growth in about 90 percent of treated patients, but most patients' response is temporary, with tumor growth resuming in six or seven months. Investigations into how this resistance emerges have suggested that the MAPK pathway gets turned back on through activation of MEK, another protein further down the pathway. Based on promising results of animal studies, the current investigation was designed to test whether inhibiting both the BRAF and MEK proteins could delay treatment resistance.

Sponsored by GlaxoSmithKline, the study by researchers at 14 sites in the U.S. and Australia tested two of the company's drugs BRAF inhibitor dabrafenib and MEK inhibitor trametinib, both oral medications currently being evaluated by the FDA as single-agent therapeutics in adult patients with BRAF-expressing malignant melanoma. Phase I testing confirmed that there were no drug-to-drug interactions between the two agents and evaluated the safety of different dose combinations. In the open-label phase II portion of the study, 162 patients were randomized into three groups that received different dose combinations: two daily 150 mg doses of dabrafenib plus one 2 mg trametinib dose, the same dabrafenib dose with a 1 mg dose of trametinib, or treatment with dabrafenib alone. Participants receiving dabrafenib alone were able to cross over to the full-dose combination treatment if their cancer resumed progression.

Treatment with both combination regimens led to a significant delay about four months longer than with dabrafenib alone in the emergence of resistance. After one year of treatment, 41 percent of those receiving full-dose combination treatment had no progression of their cancer, compared with only 9 percent of those receiving one drug. The occurrence of side effects such as skin rash and the development of squamous cell carcinoma, a less malignant skin cancer, was similar to that typically seen when only one of the two drugs is used, and some side effects were less frequent with the combination therapy.

Noting that the tested combination, now being tested in a larger Phase III study, delayed but did not prevent resistance in most participants, Flaherty says, "We need to continue focusing on resistance mechnisms occuring with this combination approach so we can better understand how to treat patients once resistance emerges or to develop other combination regimens to further prevent relapse. We also need to see if this approach could serve as an effective adjuvant therapy used following surgery to prevent recurrence. That might have the biggest impact on patients." Flaherty is an associate professor of Medicine at Harvard Medical School.

###

Jeffrey Infante, MD, of the Sarah Cannon Research Institute in Nashville is co-lead author of the NEJM paper and Jeffrey Weber, MD, PhD, of the Moffitt Cancer Center in Tampa is corresponding author. Additional participating institutions are the University of California at San Francisco, University of Colorado, Vanderbilt University, Angeles Clinic and Research Institute, University of Pennsylvania, Dana-Farber Cancer Institute, and M.D. Anderson Cancer Center in the U.S; and the Westmead Institute for Cancer Reearch and Ludwig Institute for Cancer Research in Australia.

Massachusetts General Hospital, founded in 1811, is the original and largest teaching hospital of Harvard Medical School. The MGH conducts the largest hospital-based research program in the United States, with an annual research budget of more than $750 million and major research centers in AIDS, cardiovascular research, cancer, computational and integrative biology, cutaneous biology, human genetics, medical imaging, neurodegenerative disorders, regenerative medicine, reproductive biology, systems biology, transplantation biology and photomedicine. In July 2012, MGH moved into the number one spot on the 2012-13 U.S. News & World Report list of "America's Best Hospitals."


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


[ Back to EurekAlert! ] Public release date: 29-Sep-2012
[ | E-mail | Share Share ]

Contact: Katie Marquedant
kmarquedant@partners.org
617-726-0337
Massachusetts General Hospital

Combined treatment with two drugs targeting different points in the same growth-factor pathway delayed the development of treatment resistance in patients with BRAF-positive metastatic malignant melanoma. The results of a phase I/II study of treatment with the kinase inhibitors dabrafenib and trametinib will be published in the New England Journal of Medicine and are being released online to coincide with a presentation at the European Society for Medical Oncology meeting in Vienna.

"We investigated this combination because of research we and others have conducted into the molecular underpinnings of resistance to BRAF inhibitor therapy," says Keith Flaherty, MD, of the Massachustts General Hospital (MGH) Cancer Center, lead author of the NEJM report and principal investigator of the study. "We found that adding the MEK inhibitor trametinib to BRAF inhibitor dabrafenib clearly delays the emergence of resistance. In fact, the combination was at least twice as effective as BRAF inhibition alone."

In around half of patients with metastatic melanoma, tumor growth is driven by mutations that keep the BRAF protein part of the MAPK cell growth pathway constantly activated. In recent years, drugs that inhibit BRAF activity have rapidly halted and reversed tumor growth in about 90 percent of treated patients, but most patients' response is temporary, with tumor growth resuming in six or seven months. Investigations into how this resistance emerges have suggested that the MAPK pathway gets turned back on through activation of MEK, another protein further down the pathway. Based on promising results of animal studies, the current investigation was designed to test whether inhibiting both the BRAF and MEK proteins could delay treatment resistance.

Sponsored by GlaxoSmithKline, the study by researchers at 14 sites in the U.S. and Australia tested two of the company's drugs BRAF inhibitor dabrafenib and MEK inhibitor trametinib, both oral medications currently being evaluated by the FDA as single-agent therapeutics in adult patients with BRAF-expressing malignant melanoma. Phase I testing confirmed that there were no drug-to-drug interactions between the two agents and evaluated the safety of different dose combinations. In the open-label phase II portion of the study, 162 patients were randomized into three groups that received different dose combinations: two daily 150 mg doses of dabrafenib plus one 2 mg trametinib dose, the same dabrafenib dose with a 1 mg dose of trametinib, or treatment with dabrafenib alone. Participants receiving dabrafenib alone were able to cross over to the full-dose combination treatment if their cancer resumed progression.

Treatment with both combination regimens led to a significant delay about four months longer than with dabrafenib alone in the emergence of resistance. After one year of treatment, 41 percent of those receiving full-dose combination treatment had no progression of their cancer, compared with only 9 percent of those receiving one drug. The occurrence of side effects such as skin rash and the development of squamous cell carcinoma, a less malignant skin cancer, was similar to that typically seen when only one of the two drugs is used, and some side effects were less frequent with the combination therapy.

Noting that the tested combination, now being tested in a larger Phase III study, delayed but did not prevent resistance in most participants, Flaherty says, "We need to continue focusing on resistance mechnisms occuring with this combination approach so we can better understand how to treat patients once resistance emerges or to develop other combination regimens to further prevent relapse. We also need to see if this approach could serve as an effective adjuvant therapy used following surgery to prevent recurrence. That might have the biggest impact on patients." Flaherty is an associate professor of Medicine at Harvard Medical School.

###

Jeffrey Infante, MD, of the Sarah Cannon Research Institute in Nashville is co-lead author of the NEJM paper and Jeffrey Weber, MD, PhD, of the Moffitt Cancer Center in Tampa is corresponding author. Additional participating institutions are the University of California at San Francisco, University of Colorado, Vanderbilt University, Angeles Clinic and Research Institute, University of Pennsylvania, Dana-Farber Cancer Institute, and M.D. Anderson Cancer Center in the U.S; and the Westmead Institute for Cancer Reearch and Ludwig Institute for Cancer Research in Australia.

Massachusetts General Hospital, founded in 1811, is the original and largest teaching hospital of Harvard Medical School. The MGH conducts the largest hospital-based research program in the United States, with an annual research budget of more than $750 million and major research centers in AIDS, cardiovascular research, cancer, computational and integrative biology, cutaneous biology, human genetics, medical imaging, neurodegenerative disorders, regenerative medicine, reproductive biology, systems biology, transplantation biology and photomedicine. In July 2012, MGH moved into the number one spot on the 2012-13 U.S. News & World Report list of "America's Best Hospitals."


[ Back to EurekAlert! ] [ | E-mail | Share Share ]

?


AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.


Source: http://www.eurekalert.org/pub_releases/2012-09/mgh-cot092712.php

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Mitt Romney's Tax Breaks Could Benefit Middle-Income Americans

WASHINGTON, Sept 26 (Reuters) - Much was made of multimillionaire candidate Mitt Romney's tax status when he released his 2011 tax return on Sept. 21. It showed an effective tax rate of 14.1 percent; many middle-income working Americans and the average wealthy person pay at higher rates than that.

To be sure, tax experts didn't find anything illegal or underhanded about Romney's return. In fact, he and his wife, Ann, could have had an even lower tax rate had they not left money on the table, declining to take his full deduction for charitable gifts.

But there's no need to get mad when you can get even: Middle-income taxpayers can use a lot of the same write-offs that Romney used, and they don't have to go to Switzerland or the Cayman Islands to do it.

Here are some ways that the uber-rich cut their taxes that you can use, too.

-- Invest in stocks directly. Middle-income people tend to make most of their investments via mutual funds and 401(k) plans. Those retirement funds do give you an immediate write-off of your contribution, but here's what else they do: They turn capital gains, taxed at a maximum rate of 15 percent, into ordinary income, taxed at a maximum rate of 35 percent.

And mutual funds that own portfolios full of stocks are required to distribute their taxable gains annually. Average investors who own mutual funds (outside of tax-deferred retirement funds) therefore have to pay taxes on those gains every year, whether they sell shares and withdraw money or not.

Here's the way rich folk do it: They buy shares of solid dividend-paying companies directly. They take the dividend income, also typically taxed at a 15 percent maximum. They let the stocks roll. When a share price falls, they sell the stock at a loss, and use the loss to offset other gains. When they eventually sell stocks that have long-term gains, they only pay 15 percent tax on the gain.

-- Work for yourself. Romney made his fortune and avoided big taxes by founding his own investment company, Bain Capital. But even small kitchen-table businesses confer tax breaks on their owners. If you turn your hobby into a business, work as a consultant, or provide services like childcare, lawn mowing or driving, you can cash in on some of them. Self-employed people can buy all of their equipment, supplies and services and deduct the costs; they can travel to conferences and clients and write off their trips. They can deduct the cost of their health insurance and the costs of acquiring and maintaining a home office.

More importantly, they can set aside more money on a tax-deferred basis than the typical employee. Using defined benefit retirement plans and simplified employee pensions, people who own companies can set aside substantial amounts of money for their retirement. When they leave (or simply close) their companies, they can roll that money over into tax-deferred individual retirement accounts.

-- Be charitable. As Mormons who tithe and go beyond that with big gifts to numerous nonprofits, the Romneys have substantial charitable donations that are tax deductible. They didn't even take full advantage of that write-off on their 2011 tax return; perhaps to avoid having to show a return with an effective tax rate near 10 percent, according to some reports.

But the charitable deduction is a valuable one for less well-heeled taxpayers, too. People can donate money to groups that advance their own policy stances, such as Planned Parenthood or Project Ultrasound. They can donate to their own religious organizations, or their children's private schools and camps, or their local library and symphony orchestra. And so, they can reap benefits from those donations, and still deduct them from their taxable income.

In addition, individuals can reap surprisingly substantial deductions by donating their old clothing, housewares, electronics and vehicles to charitable organizations.

And those stocks that have accumulated big gains? If you give a charity an appreciated stock, you can deduct the whole gift and you don't have to pay money on the gain. So, shares worth $1,000 that you paid $500 for can be deducted at $1,000 when you give them away to a legitimate nonprofit.

-- Help the kids. The super wealthy are widely known to use trusts and other estate-planning vehicles to transfer money to their children and grandchildren in ways that avoid taxation. But less wealthy folks can do that too. Children are allowed to receive up to $1,900 in investment income before it is taxed at their parents rate. If your children are older than 19 (or over 24 if they are full-time students) and earn $35,350 or less, their tax rate on capital gains is zero. You can give them those same appreciated stocks and they will owe no taxes on the gain.

If you decide to do that, do it this year; the zero percent capital gains tax rate expires at the end of 2012, and it's renewal is far from certain.

Also on HuffPost:

"; var coords = [-5, -72]; // display fb-bubble FloatingPrompt.embed(this, html, undefined, 'top', {fp_intersects:1, timeout_remove:2000,ignore_arrow: true, width:236, add_xy:coords, class_name: 'clear-overlay'}); });

Source: http://www.huffingtonpost.com/2012/09/27/mitt-romney-tax-breaks_n_1918700.html

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Funky Shark Penny Auction - PDF

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How to Fight Your Online Addiction and Regain Control of Your Attention [Habits]

How to Fight Your Online Addiction and Regain Control of Your AttentionHow much time do you spend each day responding to email, checking Facebook, sending and reading Tweets, aimlessly surfing your favorite websites and buying things you don't need? How much time, in other words, do you spend doing stuff online that doesn't add much value in your life, or in anyone else's? Too much, I'm going to guess.

I let it happen to me when I woke up Sunday morning, got on my laptop and started reading the New York Times. Ninety minutes later, I was still surfing from one bookmarked website to the next, vaguely aware that there were other things I wanted to do and that none of what I was taking in was very nourishing. And yet I remained narcotically glued to that screen?a baby bird with its mouth open, forever eager to be fed.

"There are few things ever dreamed of, smoked or injected that have as addictive an effect on our brains as technology," writes Stanford psychologist Kelly McGonigal in her terrific book The Willpower Instinct.

"The definitive Internet act of our times," she adds, "is a perfect metaphor for the promise of reward. We search. And we search. And we search some more...clicking that mouse...looking for the elusive reward that will finally feel like enough."

Or, as Nobel Prize winner Herbert Simon put it way back in 1978: "A wealth of information creates a poverty of attention." And retention. Taking in endless bits and bytes of information is akin to pouring water into a glass already full?in this case our severely limited working memory.

A growing body of research suggests that up to 95 percent of our behaviors occur on automatic pilot, out of habit or in reaction to an external demand or stimulus. We spend a crazily disproportionate amount of time seeking the next source of instant gratification, rather than pursuing the more challenging goals that ultimately deliver more long-term value and greater satisfaction.

It's not about summoning the strength to say "no." Each time we intentionally forgo something desirable, we deplete our already limited reservoir of will and discipline. When was the last time you resisted the seductive ping of an incoming email?

So how, then, to withstand this Pavlovian pull? And how, in turn, to take back control of your attention, so you can put it to better and richer use?

A few suggestions:

1. Lead yourself not into temptation. Instead, consciously choose times to turn off your technology entirely. The best time of all is at the start of your day, when you've typically got the most energy. Specifically, that's the best time to take on your most important and challenging tasks, without interruption, for anywhere from 30 to 90 minutes.

2. Carry a notebook with you throughout your workday. Download any ideas that come to you as quickly as possible?not just to ensure you'll remember them, but also to clear space in your working memory for whatever comes at you next. Alternatively, type the ideas into a memo pad on your smart phone.

3. Between meetings and obligations, take some time to breathe deeply?in through your nose to a count of three, out through your mouth to a count of six. In as little as one minute, you can completely clear your bloodstream of the stress hormone cortisol. You'll feel calmer, and you'll be better able to focus.

4. Take a 15- to 20-minute nap between 1 and 4 p.m.?especially on days when you've not gotten sufficient sleep and you find yourself dragging. Even a very short nap can dramatically increase your alertness and your productivity over the subsequent several hours. (This assumes, of course, than you can get your boss on board. Make the case that it'll improve your productivity.)

5. Designate and put in your calendar specific times each week to think reflectively, creatively, and/or strategically. Get up from your desk and take a walk outside, or find a comfortable, relaxing place to hang out. Leave your smart phone at your desk. The idea is to give the verbal left hemisphere of your brain a rest from its usual overload?and to rely instead during these periods on the more visual, imaginative right hemisphere of your brain. You'll know you're doing the right thing if you lose track of time.

Battling Your Online Addiction | Harvard Business Review


Tony Schwartz is the president and CEO of The Energy Project and the author of Be Excellent at Anything. Become a fan of The Energy Project on Facebook and connect with Tony on @tonyschwartz and @energy_project.

Image remixed from Mike Elliot.

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Source: http://feeds.gawker.com/~r/lifehacker/full/~3/MvkvXEO_aSM/how-to-fight-your-online-addiction-and-regain-control-of-your-attention

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The perfect shoes and foot health - Keep your mind and body sharp ...

The feet are silent hard workers of the human body. Although they don't help us see our surroundings, figure out what to say, or move stuff around, they still play a vital role. An important part of human life is going from one place to another and that is the primary responsibility of the feet.

Foot health ensures that the ability for locomotion is at its highest. This not only means that the feet are free from any structural or functional deviations, but also the maintenance this optimal state. There are many things to consider ensuring that the feet are well taken care of. One focal point of foot health is wearing the best shoes.

Everyone has experienced going barefoot during a lifetime, but the right kind foot wear is an absolute must to achieve and maintain foot health. Different activities generally require a different set of footwear. This is due to the varying levels of strain that feet are subject to during these activities. Providing the right amount of support and balance are the two main benefits of the right kind of shoes.

Different activities generally require different sets of footwear and no single pair of shoes is best-suited for all activities. However, all general activities have recommended footwear that cover the various needs of the feet during movement. Different activities like walking, running, cross-training, and sports in general place varying degrees of stress on the feet and are met with specialty kinds of shoes. Properly fitted shoes are also a key consideration in order to keep feet healthy. The right shoes with the right fit ensures that the feet are comfortable and balanced and also prevents damage to the feet resulting in corns, calluses, and plantar fasciitis.

An individual's gait type is a significant factor to determine a person's perfect pair of shoes because of individual variations in foot structures and movement patterns while walking. The general gait types are neutral pronation, overpronation, and underpronation which pose varying needs to an individual and are best accommodated by specific kinds of shoes. Maintaining effective and efficient mobility and keeping the feet healthy are two of the best benefits from wearing the perfect pair of shoes.

Source: http://idea-health-fitness.blogspot.com/2012/09/the-perfect-shoes-and-foot-health.html

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Green Blog: On Our Radar: A Miracle Crop, or a Pest?

A biofuels center is promoting mass cultivation of arundo, a fast-growing field grass that yields three times as much ethanol per acre as corn, for a proposed biofuel refinery in eastern North Carolina. But some scientists and environmentalists argue that arundo is a noxious weed that could prove as invasive as kudzu. [The News and Observer]

Citing five deadly attacks on swimmers, the Australian authorities will pre-emptively hunt and kill great white sharks along the country?s Indian Ocean coastline. The sharks had previously been a protected species in Australian waters. [Reuters]

A new study suggests that a warming climate and rising seas will help salt marshes capture more carbon dioxide from the atmosphere, possibly slowing the rate of climate change. But if seas rise too quickly, researchers say, the marshes could be overcome and lose that carbon storage capacity. [LiveScience]

The United States Geological Survey releases reams of new data from groundwater testing in a Wyoming gas field where the Environmental Protection Agency linked contamination to fracking last year. But neither agency offers any analysis, leaving it unclear whether the latest testing supports the E.P.A.?s controversial contention. [Associated Press]

Source: http://green.blogs.nytimes.com/2012/09/27/on-our-radar-a-miracle-crop-or-a-pest/?partner=rss&emc=rss

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